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In experiments of other receptor tyrosine kinases implicated in the oncogenesis of GIST, nilotinib achieved powerful and selective inhibition of PDGFRα and PDGFRβ. As is the case with imatinib, nilotinib potently inhibited the autophosphorylation of A31 cells reworked by PDGFRAEstablish a publication describing signify and variance of vancomycin